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Why We Crash Every Afternoon (and What Helps)
Jul 31, 20264 min read

Why We Crash Every Afternoon (and What Helps)

We were fine this morning. Focused, even. Then somewhere after lunch it hits: our eyes get heavy, our thoughts go slow, and getting anything done feels like walking through a swamp. This is the afternoon crash, and almost all of us get it. It is not a sign we are lazy or out of shape. It is one of the most normal things our bodies do.

Why It Happens

The afternoon crash is not one thing going wrong. It is a few normal rhythms in our bodies lining up at the same time of day. Once we see what they are, the whole thing makes more sense.

The body clock has an afternoon dip

Our bodies run on an internal clock, and it does more than tell us when to sleep. It also raises and lowers our energy at set times of day. One of those lows lands in the early afternoon. It arrives whether or not we ate lunch.¹ It is natural, and one of the biggest reasons we feel so wiped by mid-afternoon.

A sleep signal builds up all day

From the moment we wake up, a molecule called adenosine starts to build up in the brain. This is what makes us feel sleepy as the day goes on.² The longer we are awake, the more we collect, and the sleepier we feel. By the afternoon we have collected so much that what was easy in the morning now takes forever.

A heavy lunch adds to it

What we eat matters too. A heavy lunch, especially a carb-heavy one, makes the dip worse.¹ The timing is the catch. It lands right on top of the afternoon dip, until we barely have the energy for anything. So we go looking for a way through, and for most of us that means another coffee.

Which is where coffee comes in

Caffeine helps for a while, but it has its own timing issues, both when it wears off and how late we drink it. That is a whole topic on its own, and we get into it in Is Rhodiola a Better Alternative to Caffeine.

Taking the Edge Off

A few small things take the edge off.


Stepping away from the screen for a few minutes helps, especially if we can get outside. Daylight nudges the internal clock,³ and moving around, even a slow walk, shakes off some of the heaviness.

There is also the snack urge. When we are short on sleep, we get hungrier and reach for more snacks, especially carb-heavy ones.⁴ That craving we feel is not weakness, it's our tired body looking for quick fuel. This works for a bit, but leaves us in the same exact spot.
These will not erase the crash, but they make it easier to get through.

The small stuff only goes so far, though. If the tiredness underneath is the bigger issue, some of us look to a supplement for help. Salidroside is the one worth knowing about. It won't fix the dip. But it's studied for that deeper, run-down kind of tired that turns an ordinary afternoon into a wall.

Where Salidroside Comes In

Salidroside is the main active compound in rhodiola,⁵ a plant used for a long time to fight fatigue.

In lab studies, salidroside switches on AMPK, the master switch our cells use to make energy. It also acts as an antioxidant, mopping up the cellular stress that leaves us run-down.⁶ That is why it is studied for the deeper kind of tired, not the afternoon dip, but the background fatigue that makes the dip hit harder.

If you want the full picture, we get into it in One Compound, A lot of Claims.
The evidence in people is still early. One small trial found that people taking salidroside held steadier under stress than a placebo group.⁵ It is a start, not proof.

So What Do We Do?

The afternoon crash is mostly our bodies doing what they are built to do. We cannot switch off the body clock or the build-up of adenosine. But a few small habits take the edge off, and we can support the tiredness underneath.


Salidroside is the active behind rhodiola's long history with fatigue. It will not stop the dip. It helps with the run-down feeling that sits under it.

Most rhodiola supplements are whole-plant extracts. They often give us only about 15 mg of salidroside, and the amount is hard to pin down. Which is why we made our 98% High-Purity Salidroside with a set 60 mg every time. That is the same dose used in the human trial of salidroside on its own.⁵


These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure, or prevent any disease.

References

  1. Monk TH. The post-lunch dip in performance. Clin Sports Med. 2005;24(2):e15-e23, xi-xii. doi:10.1016/j.csm.2004.12.002
  2. Reichert CF, Maire M, Schmidt C, Cajochen C. Sleep-Wake Regulation and Its Impact on Working Memory Performance: The Role of Adenosine. Biology (Basel). 2016;5(1):11. doi:10.3390/biology5010011
  3. Münch M, Bromundt V. Light and chronobiology: implications for health and disease. Dialogues Clin Neurosci. 2012;14(4):448-453. doi:10.31887/DCNS.2012.14.4/mmuench
  4. Tajiri E, Yoshimura E, Tobina T, et al. Effects of sleep restriction on food intake and appetite under free-living conditions: A randomized crossover trial. Appetite. 2023;189:106998. doi:10.1016/j.appet.2023.106998
  5. Schwarz NA, Stratton MT, Colquhoun RJ, et al. Salidroside and exercise performance in healthy active young adults: an exploratory, randomized, double-blind, placebo-controlled study. J Int Soc Sports Nutr. 2024;21(1):2433744. doi:10.1080/15502783.2024.2433744
  6. Ju L, Wen X, Wang C, et al. Salidroside, A Natural Antioxidant, Improves β-Cell Survival and Function via Activating AMPK Pathway. Front Pharmacol. 2017;8:749. doi:10.3389/fphar.2017.00749
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Amy Qin, PhD, RD, CDCES, Nutrition Scientist at Neumina

Amy Qin is a Nutrition Scientist at Neumina with training in both nutrition research and clinical care. She received her PhD in Nutrition and Metabolism from the University of Wisconsin-Madison and completed clinical training at Stanford Hospital and UCSF Benioff Children's Hospital.

Her work focuses on applying nutrition science to metabolism, aging, and chronic disease management in ways that are practical and personalized.